Surreal illustration of a pregnant woman surrounded by glowing IL-27 molecules.

Preeclampsia and IL-27: New Insights into Maternal Health

"Discover the crucial link between IL-27 levels and preeclampsia, a severe pregnancy complication, and how it could impact diagnosis and treatment."


Preeclampsia (PE) is a pregnancy-specific syndrome characterized by hypertension and proteinuria, typically arising after 20 weeks of gestation. Affecting 2-8% of pregnancies worldwide, PE remains a significant contributor to maternal and neonatal morbidity and mortality. Despite ongoing research, the precise causes and mechanisms underlying PE are not fully understood, highlighting the urgent need for improved diagnostic and therapeutic strategies.

Recent research has increasingly focused on the role of the immune system and inflammation in the development of PE. Rather than a controlled inflammatory response, PE is believed to involve an exaggerated maternal immune reaction to placental stimuli. This response is characterized by an imbalance in T helper cells (Th1 and Th2), leading to a cascade of inflammatory cytokine production. Cytokines like IL-1β, IL-6, IL-8, IL-12, TNF-α, and IFN-γ are often elevated in PE, while protective cytokines like IL-4, IL-5, and IL-10 are reduced, further disrupting the delicate balance required for a healthy pregnancy.

Interleukin-27 (IL-27), a member of the IL-12 cytokine family, has emerged as a key player in immune regulation. Composed of two subunits, EBI3 and IL-27p28, IL-27 is primarily produced by antigen-presenting cells (APCs). Its receptor, WSX-1/TCCR, is widely expressed on various immune cells, including T cells, B cells, NK cells, and even non-immune cells like endothelial cells and trophoblasts. Given its diverse effects on immune responses, researchers have begun to investigate the role of IL-27 in PE, aiming to uncover potential diagnostic and therapeutic targets.

Unveiling the Link: IL-27 and Preeclampsia Severity

Surreal illustration of a pregnant woman surrounded by glowing IL-27 molecules.

A recent study published in Cytokine delved into the relationship between maternal circulating IL-27 levels and preeclampsia, offering new insights into the disease's severity and potential diagnostic markers. The research, led by Danial Jahantigh and colleagues, involved 56 preeclamptic women, 21 healthy pregnant women, and 20 healthy non-pregnant women. The study measured IL-27 serum levels using ELISA assays to determine if any correlation existed between IL-27 levels and the presence or severity of preeclampsia.

The study revealed several significant findings. First, IL-27 serum levels were elevated in both healthy pregnant women and preeclamptic women compared to non-pregnant women. However, the increase was significantly more pronounced in the preeclamptic group, suggesting a potential link between IL-27 and the disease. Furthermore, the researchers observed a significant difference in IL-27 levels between the preeclamptic group and the healthy pregnant controls, indicating that elevated IL-27 might be a marker for PE.

  • Severity Matters: IL-27 levels were notably higher in women with severe preeclampsia compared to those with mild preeclampsia, indicating that IL-27 levels could correlate with the severity of the disease.
  • Timing is Key: Women with early-onset severe preeclampsia had significantly different IL-27 levels than gestation-matched healthy pregnancies, suggesting IL-27 could be involved in the early stages of severe PE.
  • Fetal Growth: Although the study did not find a significant relationship between IL-27 levels and fetal growth restriction (FGR), the potential impact of IL-27 on placental function and fetal development warrants further investigation.
These findings highlight the potential of IL-27 as a biomarker for preeclampsia severity. While further research is needed to fully understand the role of IL-27 in the pathogenesis of PE, the current study provides a compelling foundation for future investigations into diagnostic and therapeutic strategies.

Future Directions: IL-27 as a Therapeutic Target

The findings from this study open up new avenues for exploring IL-27 as a potential therapeutic target for preeclampsia. Understanding how IL-27 contributes to the inflammatory processes in PE could lead to the development of targeted therapies to modulate its activity, potentially reducing the severity of the disease and improving outcomes for both mother and child. Further research is crucial to elucidate the precise mechanisms by which IL-27 influences the pathogenesis of PE and to evaluate its potential as a diagnostic and therapeutic tool.

About this Article -

This article was crafted using a human-AI hybrid and collaborative approach. AI assisted our team with initial drafting, research insights, identifying key questions, and image generation. Our human editors guided topic selection, defined the angle, structured the content, ensured factual accuracy and relevance, refined the tone, and conducted thorough editing to deliver helpful, high-quality information.See our About page for more information.

This article is based on research published under:

DOI-LINK: 10.1016/j.cyto.2017.08.012, Alternate LINK

Title: Association Between Maternal Circulating Il-27 Levels And Preeclampsia

Subject: Molecular Biology

Journal: Cytokine

Publisher: Elsevier BV

Authors: Danial Jahantigh, Mahdieh Mousavi, Forough Forghani, Mohammad Reza Javan, Samaneh Movahedinia, Mahnaz Rezaei

Published: 2018-02-01

Everything You Need To Know

1

What is preeclampsia, and what are the key factors contributing to its development during pregnancy?

Preeclampsia (PE) is a pregnancy-specific syndrome characterized by hypertension and proteinuria, typically arising after 20 weeks of gestation. It significantly contributes to maternal and neonatal morbidity and mortality. While the precise causes are not fully understood, research suggests an exaggerated maternal immune reaction to placental stimuli. This involves an imbalance in T helper cells (Th1 and Th2) and the production of inflammatory cytokines, which disrupts the balance needed for a healthy pregnancy.

2

What is Interleukin-27 (IL-27), and how does it function within the immune system?

Interleukin-27 (IL-27) is a member of the IL-12 cytokine family composed of two subunits, EBI3 and IL-27p28, and is primarily produced by antigen-presenting cells (APCs). Its receptor, WSX-1/TCCR, is expressed on various immune cells, including T cells, B cells, NK cells, and even non-immune cells like endothelial cells and trophoblasts. IL-27 is a key player in immune regulation.

3

How are Interleukin-27 (IL-27) levels different in pregnant women with and without preeclampsia, and what does this suggest about the role of IL-27?

Research indicates that IL-27 serum levels are elevated in both healthy pregnant women and preeclamptic women compared to non-pregnant women. However, the increase is significantly more pronounced in the preeclamptic group. Furthermore, IL-27 levels were notably higher in women with severe preeclampsia compared to those with mild preeclampsia. Women with early-onset severe preeclampsia also had significantly different IL-27 levels than gestation-matched healthy pregnancies.

4

Does Interleukin-27 (IL-27) impact fetal growth in pregnancies complicated by preeclampsia, and what further research is needed in this area?

While this research did not find a significant relationship between IL-27 levels and fetal growth restriction (FGR), it is an area that needs further study. The potential impact of IL-27 on placental function and fetal development requires further investigation to determine if IL-27 plays a role in fetal growth problems associated with preeclampsia. Understanding this connection could provide new strategies for managing preeclampsia and improving fetal outcomes.

5

How might Interleukin-27 (IL-27) be used as a therapeutic target for preeclampsia, and what future research is needed to explore this potential?

Targeted therapies could be developed to modulate the activity of IL-27, potentially reducing the severity of preeclampsia and improving outcomes for both mother and child. Future studies should focus on the precise mechanisms by which IL-27 influences the pathogenesis of PE and evaluate its potential as a diagnostic and therapeutic tool. Further research could explore drugs or other interventions that specifically target the IL-27 pathway to manage the inflammatory response in preeclampsia.

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